Understand the source comparison
How to Use Peptides for Ulcer Healing: Peptide Comparison
Before selecting a peptide for ulcer healing, compare the mechanisms, dosing standards, and evidence base for the most-researched compounds. BPC-157 Stimulates VEGF and fibroblast growth factor; promotes angiogenesis in damaged mucosa 200–500 mcg daily subcuta
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This page preserves a source comparison for education. It does not add a rating, recommendation or clinical judgment.
- Before selecting a peptide for ulcer healing, compare the mechanisms, dosing standards, and evidence base for the most-researched compounds.
- BPC-157
- Stimulates VEGF and fibroblast growth factor; promotes angiogenesis in damaged mucosa
- 200–500 mcg daily subcutaneous
- 40+ animal studies; reduces gastric ulcer area by 80–90% in 14 days
- Most documented peptide for gastric ulcer healing. Consistent results across rat, mouse, and rabbit models
- TB-500 (Thymosin Beta-4)
- Upregulates actin polymerisation; accelerates cell migration and wound closure
- 2–5mg twice weekly subcutaneous
- Primarily studied in musculoskeletal injury; limited gastric-specific data
- Broader tissue repair peptide with indirect ulcer benefit through systemic healing pathways
- KPV 5MG
- Anti-inflammatory tripeptide; inhibits TNF-alpha and reduces inflammatory cytokines in gut tissue
- 500 mcg–1mg daily
- Emerging research in IBD models; shows promise for inflammatory ulcers
- Best suited for ulcers with inflammatory component. Less direct angiogenic effect than BPC-157
- Pentadecapeptide BPC-157 Fragment
- Isolated C-terminal fragment retaining cytoprotective properties
- 100–300 mcg daily
- Limited independent research; mostly studied as part of full BPC-157
- Theoretical advantage in stability but insufficient evidence to recommend over full BPC-157
- BPC-157 has the strongest evidence base for gastric and duodenal ulcer healing. TB-500 works systemically but lacks gastric-specific validation. KPV is anti-inflammatory rather than angiogenic. It's a supporting compound, not a primary ulcer-healing agent.