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Peptides Help with Ulcer Healing: Evidence Comparison
BPC-157 VEGF/FGF pathway activation; angiogenesis; epithelial migration 58% faster ulcer closure at 14 days vs control (J Physiol Pharmacol 2019) 10 mcg/kg daily (subcutaneous) Strongest evidence base for gastric ulcer healing. Direct growth factor receptor ag
No winner is assigned.
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- BPC-157
- VEGF/FGF pathway activation; angiogenesis; epithelial migration
- 58% faster ulcer closure at 14 days vs control (J Physiol Pharmacol 2019)
- 10 mcg/kg daily (subcutaneous)
- Strongest evidence base for gastric ulcer healing. Direct growth factor receptor agonism
- Thymosin Beta-4
- Actin polymerization; epithelial cell migration; anti-apoptotic signaling
- 63% reduction in ulcer diameter at day 10 (Wound Repair Regen 2020)
- 5–10 mg weekly (subcutaneous)
- Complementary to BPC-157. Targets cell migration rather than angiogenesis
- KPV Tripeptide
- Anti-inflammatory; reduces TNF-alpha and IL-6; lowers mucosal oxidative stress
- 47% reduction in gastric inflammation markers within 72 hours (Inflamm Bowel Dis 2021)
- 500 mcg–1 mg daily (oral or subcutaneous)
- Adjunctive benefit by reducing inflammatory barriers to healing
- Collagen Peptides (Generic)
- Provides amino acids (glycine, proline, hydroxyproline)
- No direct evidence for gastric ulcer healing. Lacks receptor binding specificity
- 10–20 g daily (oral)
- Structural protein building blocks but no documented growth factor activity