Founder of Stemcode Peptides | Jeff S.

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Jeff S.

Founder & CEO of Stemcode Peptides

Building Stemcode

Under Jeff's leadership, Stemcode Peptides has grown from a focused startup into a trusted name in the research peptide space. Key milestones include:

Establishing partnerships with ISO-certified third-party laboratories for independent batch testing

Developing proprietary nasal spray formulations with optimized pH, preservatives, and stability profiles

Building a catalog of 24+ research peptides across nasal sprays, tablets, and topical formats

Implementing cold-chain shipping protocols to ensure product integrity from warehouse to lab

Creating a publicly accessible COA library for full research transparency

Research-First Philosophy

Jeff's approach to business mirrors the scientific method itself: make claims only when backed by data. Every Stemcode product ships with a Certificate of Analysis documenting HPLC purity, mass spectrometry identity confirmation, and batch-specific results. There are no proprietary blends, no undisclosed ingredients, and no ambiguity about what's in each product.

"Researchers need to trust their materials," Jeff says. "If you can't verify what you're working with, you can't trust your results. We built Stemcode to solve that problem."

Compliance & Integrity

Stemcode Peptides operates in strict compliance with research-use-only standards. All products are manufactured, labeled, and sold exclusively for in-vitro research, laboratory experimentation, and educational purposes. Jeff has been instrumental in establishing the company's internal Code of Conduct and WAAVE compliance framework, ensuring that every aspect of Stemcode's operations meets or exceeds industry standards.

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Research context

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Published, peer-reviewed studies

A nootropic adrenocorticotropin analog 4-10-semax (15 years experience in its design and study) Semax in prevention of disease progress and development of exacerbations in patients with cerebrovascular insufficiency Semax, an analog of ACTH(4-10) with cognitive effects, regulates BDNF and trkB expression in the rat hippocampus Study demonstrating Semax-induced upregulation of BDNF and its receptor TrkB in rat hippocampus and cortex, supporting neuroprotective mechanisms.

Source: stemcodepeptides.com ↗

Peer-Reviewed Studies

Our compounds are backed by published, peer-reviewed scientific literature. Click any compound to explore the research. BPC-157 is a 15-amino acid peptide fragment derived from human gastric juice. It has been extensively studied in pre-clinical models across gastrointestinal, musculoskeletal, and neurological research contexts. Curr Pharm Des (2018). “BPC 157 and Standard Angiogenic Growth Factors. Gastrointestinal Tract Healing, Lessons from Tendon, Ligament, Muscle and Bone Healing.” View source ↗ J Physiol Paris (1997). “BPC 157's effect on healing.” View source ↗ Vasc Pharmacol (2018). “Rat inferior caval vein (ICV) ligature and particular new insights with the stable gastric pentadecapeptide BPC 157.” View source ↗ Molecules (2014). “Pentadecapeptide BPC 157 enhances the growth hormone receptor expression in tendon fibroblasts.” View source ↗ World J Gastroenterol (2017). “Stable gastric pentadecapeptide BPC 157 in the treatment of colitis and ischemia and reperfusion in rats: New insights.” View source ↗ GHK-Cu is a naturally occurring tripeptide-copper complex with documented effects on gene expression, collagen synthesis, and extracellular matrix remodeling in published literature. Int J Mol Sci (2018). “Regenerative and protective actions of the GHK-Cu peptide in the light of new gene data.” View source ↗ BioMed Res Int (2015). “GHK peptide as a natural modulator of multiple cellular pathways in skin regeneration.” View source ↗ Cosmetics (2012). “GHK-Cu may prevent oxidative stress in skin by regulating copper and modifying expression of numerous antioxidant genes.” View source ↗ J Pept Sci (2008). “The human tri-peptide GHK and tissue remodeling.” View source ↗ TB-500 is a synthetic fragment of Thymosin Beta-4, a naturally occurring 43-amino acid peptide. It is the primary actin-sequestering protein in mammalian cells and has been studied for its role in cell migration, angiogenesis, and tissue repair. Expert Opin Biol Ther (2012). “Thymosin β4: a multi-functional regenerative peptide. Basic properties and clinical applications.” View source ↗ Ann N Y Acad Sci (2010). “Thymosin beta4 and corneal wound healing: visions of the future.” View source ↗ Nature (2004). “Thymosin beta4 activates integrin-linked kinase and promotes cardiac cell migration, survival and cardiac repair.” View source ↗ Semax is a synthetic heptapeptide derived from the ACTH(4-7) fragment with a Pro-Gly-Pro C-terminal extension. Developed at the Institute of Molecular Genetics in Moscow, it has been studied for nootropic and neuroprotective properties. Zh Vyssh Nerv Deiat Im I P Pavlova (1997). “A nootropic adrenocorticotropin analog 4-10-semax (15 years experience in its design and study).” View source ↗ Zh Nevrol Psikhiatr Im S S Korsakova (2005). “Semax in prevention of disease progress and development of exacerbations in patients with cerebrovascular insufficiency.” View source ↗ Brain Res (2006). “Semax, an analog of ACTH(4-10) with cognitive effects, regulates BDNF and trkB expression in the rat hippocampus.” View source ↗ Epitalon (Ala-Glu-Asp-Gly) is a synthetic tetrapeptide studied by Prof. Vladimir Khavinson at the Saint Petersburg Institute of Bioregulation and Gerontology for its effects on telomerase activity and pineal gland function. Bull Exp Biol Med (2003). “Epithalon peptide induces telomerase activity and telomere elongation in human somatic cells.” View source ↗ Biogerontology (2003). “Effect of Epitalon on biomarkers of aging, life span and spontaneous tumor incidence in female Swiss-derived SHR mice.” View source ↗ MOTS-c is a 16-amino acid peptide encoded within the mitochondrial 12S rRNA gene. Discovered by Dr. Changhan Lee at USC, it has been studied for its role in metabolic homeostasis and exercise physiology. Cell Metab (2015). “The mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and reduces obesity and insulin resistance.” View source ↗ Nat Commun (2021). “MOTS-c is an exercise-induced mitochondrial-encoded regulator of age-dependent physical decline and muscle homeostasis.” View source ↗ Research Use Only. All references are to published peer-reviewed literature. No therapeutic claims are made or implied. Products are for laboratory research purposes only.

Source: stemcodepeptides.com ↗

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